Sustanon 250 product was developed by Organon as an ideal HRT (Hormone Replacement Therapy) solution, and it was thought at the time that the different esters would be able to provide a constant release of Testosterone over a months time. Sustanon is a blend of different estered testosterones (4 of them): testosterone propionate - 30 mg, testosterone phenylpropionate - 60 mg, testosterone isocaproate - 60mg, and testosterone decanoate -100 mg.

This drug was highly sought after as a "superior" version of testosterone in the late 80´s and through the mid 90´s. No doubt this is partly due to the very nice write-up Dan Duchaine gave it in his newsletters. However, lets keep in mind that this drug was designed for convenience, not athletics or bodybuilding. The advantage to this drug, according to the manufacturer, is that it can be injected once a month, and the different esters would provide different timed releases over that month, and the patient would therefore only need to visit the doctor once a month for his shot. For athletes or bodybuilders (who routinely use between half a gram and a gram of testosterone per week), this product is really no better than any other form of injectable testosterone.

Lately, it seems that this product has fallen out of favor with Steroid members, as many feel that the inclusion of the Propionate and phenylpropionate estered forms of testosterone in this blend would necessitate shooting every other day. This stems from the fact that testosterone propionate would be shot every other day at least, and testosterone phenylpropionate would generally be shot every third day.

Sustanon will do exactly what other forms of testosterone will do:
Testosterone will cause both muscle growth as well as fat loss. It sends a message to muscle cells to store more contractile protein (called actin and myosin), thus making your muscles grow. It also protects your muscles from catabolic (muscle wasting) glucocorticoid hormones(1). Thus it is often said that testosterone is not only anabolic, but it is strongly catabolic. Not only does it cause an increase in size of the muscle fibres (hyperfascia) but it also can change the appearance and the actual number of muscle fibres (Hyperplasia)(2). Testosterone has the ability to increase erythropoiesis (red blood cell production) in your kidneys(4), and a higher Red Blood Cell (RBC) count may improve endurance by producing more highly oxygenated blood. More RBCs can also improve recovery from strenuous physical activity. Agression levels often rise dramatically with the use of any exogenous testosterone (3). Testosterone improves muscle contraction by increasing the number of motor neutrons in muscle(5) and improves neuromuscular transmission(6). It also promotes glycogen synthesis(7)
And, since Sustanon is simply a form of (well actually 4 forms of) testosterone, we also know that administration of this compound will produce a dose respondant curve. (10)A what? Yeah...basically a "dose respondant curve" is the fancy way of saying "the more you take, the bigger you get..."
This is true of Sustanon as well as for every form of testosterone, up to a point.

Unfortunately, Sustanon will also do all of the bad things that any form of testosterone is known for:
It will convert to the female hormone estrogen (via a mechanism known as aromatization) by the (you guessed it) aromatize enzyme. Excessive estrogen can lead to unwanted side effects, such as acne, the growth of breast tissue (gynecomastia), fat gain and reduced fat breakdown, loss of sex drive, testicular shrinkage and water retention. Water retention can increase blood pressure weakening blood vessels over time. Unfortunately, this isn´t all it does& it can also interact with the 5 alpha-reductase enzyme. This interaction converts the testosterone to Dihydro-testosterone (DHT), a more androgenic form of the parent hormone. DHT has a high binding affinity to the tissues of the scalp resulting in hair loss in loss in users who suffer from male pattern baldness. DHT can affect the prostate as well, making it larger. This swelling can cause the gland to press against the bladder causing urinary problems. Drugs called 5alpha-reductase inhibitors can prevent these symptoms without blocking testosterone´s anabolic effects.(8) Higher dosages of test can also negatively impact cholesterol, lowering HDL(9). Testosterone is probably the safest steroid around, but it can´t be taken lightly, and Sustanon is no different.

The principal drawback to Sustanon is it´s cost. It can cost between $5 an ampule and $12 an ampule. Compared with Omnadren, Testoviron, or even Sten (other testosterone products featuring various blends of Testosterone), the cost makes it prohibitive. An equal amount of an of the aforementioned products can be had for less than half the average cost of an amp of Sustanon. Sustanon, therefore, is no better or worse than any other form of testosterone... if the price is right.

17b-hydroxy-4-androsten-3-one Testosterone base + 4 different esters Propionate, Phenylpropionate, Isocaproate, Decanoate Formula (base): C27 H40 O3 Molecular Weight (base): 288.429 Molecular Weight, Esters: Propionate: 362.5082 Phenylpropionate: 438.6058 Isocaproate: 404.5886 Decanoate: 460.6958 Formula (base): C19 H28 O2 Melting Point (base): 155 Manufacturer: Organon Effective Dose (Men): 500-2000mg/ week Effective Dose (Women): Not recommended Active life: Up to 3 weeks Detection Time: 3+ months Anabolic/Androgenic ratio:100/100

Omnadren 250
Omnadren 250 is a combination of the 4 separate test esters listed above. Older versions of the drug list the final two esters as ´isohexanoate´ and ´hexanoate.´ However, it should be noted that hexanoate is simply another word for caproate so the drug´s esters have not actually been modified. Most commonly, people will correlate Omnadren 250 with its cousin Sustanon 250, since they are both a blend of 4 test esters. The only difference between the two lies in the last  and most concentrated- ester. Whilst Omnadren contains the caproate ester, Sustanon contains the decanoate ester in the same concentration. Really, except for price, there´s no difference& and price-wise, you´re going to be paying ½ as much for Omnadren as you would for Sustanon (or $3-4usd/amp). This is a very nice price, and for that reason, I typically advise people to purchase Omnadren over Sustanon, if their source carries it.

It is also not uncommon to hear people refer to Omnadren as a superior version of testosterone since it boasts 4 esters instead of 1 (or none). This should be taken with a grain of salt. All testosterone´s produce very similar effects while the ester simply delays the release of the compound into the body which has two immediate consequences. The first, being less important, is injection frequency. This has become a hotly debated issue recently& on the one side are those who advocate injections only once or twice per week. Frequently their arguments are supported with cycle results which have yielded ´good gains.´ On the other side  and perhaps the more scientific side- are those who advocate injections at least every other day (EOD) or ever day (ED). One has only to glance at the ester constitution in Omnadren to understand why this may be. Such small concentrations of the shorter esters (propionate and phenylpropionate) are rendered practically useless when Omnadren is injected once or twice per week. Furthermore, when injecting only a few times per week the ´peaks and valleys´ of concentration in the blood are not desirable. We want our blood concentration of the drugs to be as high as they can be  relative to dose- as long as they can be. Obviously, this is not the case when fast acting esters are introduced and subsequently dissipated before another injection is given.

As the longest ester in Omnadren (caproate) is slightly faster acting than the longest ester in Sustanon (decanoate), users will notice an increase in their testosterone levels sooner with Omnadren than with Sustanon. This has a few consequences which we shall examine now. First of all, since testosterone aromatizes (converts) to estrogen, a buildup of this female hormone will occur more rapidly. With estrogen increase follows the inevitability of increased water retention. This is significant for 3 reasons: First, the user´s strength will increase. Secondly, the user´s size will increase, and finally, definition in the muscles will begin to dissipate. As an obvious result, Omnadren is typically used more for bulking than cutting. The extent of these effects are highly dictated by the user´s diet and training habits, although it is also easily controlled with the proper use of anti-estrogen drugs such as Nolvadex, Armidex, Proviron, and a myriad of others.

As I previously stated, testosterone is a highly anabolic and androgenic hormone, it has an anabolic (muscle building) rating of 100, making it a good drug to use if one is in pursuit of more size and strength. And if you aren´t in pursuit of more size and strength, then why would you be reading this, right? Well, let´s get on with it and look at exactly what makes testosterone a good mass builder. Firstly, testosterone promotes nitrogen retention in the muscle (6) the more nitrogen the muscles holds the more protein the muscle stores. Testosterone can also increase the levels of the highly anabolic hormone, IGF-1, in muscle tissue (7)(9). Even the aromatized part of testosterone that turns into estrogen may increase levels of IGF, and sensitivity to it. Testosterone´s actions come mostly from it´s binding to the androgen receptor to promote A.R dependant mechanisms for both muscle gain and fat loss (5). Thankfully, it also significantly increases the concentrations of the A. R in cells critical for muscle repair and growth and A.R in muscle (8 ). Testosterone induces changes in shape, size and also can change the appearance and the number of muscle fibers (7). Androgens like the testosterone(s) found in Omnadren can protect your hard earned muscle from the catabolic hormones (8), whether those hormones occur from exercise or other stress.

Omnadren Side EffectsThere are strong androgenic side effects, which are pronounced with Omnadren (as with all testosterones). Oily skin, acne, increased body/facial hair, and depending on the individual an increase in aggressiveness can occur. Omnadren can also be hard on the hairline. This is partly due to the conversion of the testosterone into dihydrotestosterone (DHT). Test is converted to DHT via the 5-alpha reductase enzyme. While DHT is more potent than test at the androgen receptor (the double bond is removed from the carbon4-carbon5 bond and replaced with a hydrogen atom on each) and is responsible for some growth. It can also cause some negative side effects as well. Testosterone, because of this bond is actually much more anabolic, in practical terms, because it For example: DHT formation in the scalp is suspected of causing/expediting male pattern baldness. To possibly combat this, one can use finasteride (Proscar®). This drug will inhibit the conversion of testosterone to DHT but many users will report that since DHT is more potent at the androgen receptor than test, gains in muscle mass as well as strength will diminish. On the other hand, a lack of DHT caused by blocking 5-AR can sometimes cause gynocomastia (4)(5).

Omnadren Cycles
Typically cycles which contain Omnadren 250 will be around 12-16weeks. The idea is that it will take at least 2 weeks for the compound to become fully ´active´ in the body, and most users will report an additional 1-3 weeks until the effects of Omnadren are truly felt. As a result, gains from Omnadren are not typically noticed for about 1 month after the first injection. What most people mean by this is that although the actual drug is already active, gains aren´t realized immediately. The majority of users will supplement a fast acting oral drug such as Dianabol or Anadrol in the first 4 weeks of a cycle which is thought of as a ´kickstart´ until the effects of the Omnadren are fully felt. As mentioned above a typical weekly dose of Omnadren can range from 500mg-1000mg per week. Those who are new to steroids and cycling should generally start with a minimal dose so as to better judge how their own bodies will react to the synthetic testosterone. I´d suggest beginners stick with 2 amps per week, if they´re inclined to use this preparation.
Omnadren has always been manufactured by Polfa© which has since changed their name to Jelfa©. The company is based in Poland, and as one might obviously conclude, the availability and price of Omnadren 250 there is much different than many other places. Often, fake Sustanon in the 80´s would actually turn out to be Omnadren, which was much less highly prized (nonsensically).

Omnadren (Testosterone) Profile1 milliliter of Omnadren 250 contains: 30mg testosterone propionate 60mg testosterone phenylpropionate 60mg testosterone isocaproate 100mg testosterone caproate
[4-androsten-3-one-17beta-ol] 17beta-hydroxy-androst-4-en-3-one Molecular Weight (of Base): 288.429 Molecular Weight (esters) Propionate: 362.5082 Phenylpropionate: 438.6058 Isocaproate: 404.5886 Caproate:116.16 Formula (of Base): C19 H28 O2 Formula (esters) Propionate:C3 H6 O2 Phenylpropionate:C9 H10 O2 Isocaproate:C6 H12 O2 Caproate:C16 H12 O2 Melting Point: 154-155C Manufacturer: Jelfa Effective dose: 250-1,000mgs/week Active Life: 10 days Detection Time: 3 months Anabolic/Androgenic Ratio: 100:100
References:
Hypothalamic sites of action for testosterone, dihydrotestosterone, and estrogen in the regulation of luteinizing hormone secretion in male sheep. Endocrinology. 1997 Sep;138(9):3686-94. Inhibition of LH Secretion by Localized Administration of Estrogen, but not Dihydrotestosterone, Is Enhanced in the Ventromedial Hypothalamus During Feed Restriction in the Young Wether. Biol Reprod. 2005 Jun 22; [Epub ahead of print] Crystalline dihydrotestosterone implants in the lateral septum of male rats. A positive effect on LH and FSH. Endocr Res. 2001 Feb-May;27(1-2):35-40. Significant role of 5 alpha-reductase on feedback effects of androgen in rat anterior pituitary cells demonstrated with a nonsteroidal 5 alpha-reductase inhibitor ONO-3805. J Androl. 1994 Nov-Dec;15(6):521-7. Case report: finasteride-induced gynecomastia in a 62-year-old man. Am J Med Sci. 1995 Jun;309(6):322-5. J Clin Endocrinol Metab. 1997 Feb;82(2):407-13. Am J Physiol Endocrinol Metab. 2002 Mar;282(3):E601-7. Curr Opin Clin Nutr Metab Care. 2004 May;7(3):271-7. Comparison of effects of the rise in serum testosterone by raloxifene and oral testosterone on serum insulin-like growth factor-1 and insulin-like growth factor binding protein-3. Maturitas. 2005 Jul 16;51(3):286-93.