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Clomid ® is the commonly referenced brand name for the drug clomiphene citrate. It is not an anabolic steroid, but a prescription drug generally prescribed to women as a fertility aid. This is due to the fact that clomiphene citrate shows a pronounced ability to stimulate ovulation. This is accomplished by blocking/minimizing the effects of estrogen in the body. To be more specific Clomid is chemically a synthetic estrogen with both agonist/antagonist properties, and is very similar in structure and action to Nolvadex. In certain target tissues it can block the ability of estrogen to bind with its corresponding receptor. Its clinical use is therefore to oppose the negative feedback of estrogens on the hypothalamic-pituitary-ovarian axis, which enhances the release of LH and FSH. This of course can help to induce ovulation.

For athletic purposes, Clomid does not offer a tremendous benefit to women. In men however, the elevation in both follicle stimulating hormone and (primarily) luteinizing hormone will cause natural testosterone production to increase. This effect is especially beneficial to the athlete at the conclusion of a steroid cycle when endogenous testosterone levels are depressed. If endogenous testosterone levels are not brought beck to normal, a dramatic loss in size and strength is likely to occur once the anabolics have been removed. This is due to the fact that without testosterone (or other androgens), the catabolic hormone cortisol becomes the dominant force affecting muscle protein synthesis (quickly bringing about a catabolic metabolism). Often referred to as the post-steroid crash, it can quickly eat up much of your newly acquired muscle. Clomid can play a crucial role in preventing this crash in athletic performance. As for women, the only real use for Clomid is the possible management of endogenous estrogen levels near contest time. This can increase fat loss and muscularity, particularly in female trouble areas such as this hips and thighs. Clomid however often produces troubling side effects in women (discussed below), and is likewise not in very high demand among this group of athletes.

Male users generally find that a daily intake of 50-100 mg (1-2 tablets) over a four to six week period will bring testosterone production back to an acceptable level. A very common regime of dosing is; 300 md/day 1, 100 mg/day for days 2-11, and 50 mg/day for days 12-21. This raise in testosterone should occur slowly but evenly throughout the period of intake. Since an immediate boost in testosterone is often desirable, many prefer to combine Clomid with HCG (Human Chorionic Gonadotropin) for the first week or two after the steroids have been removed. The kick-start from HCG also helps to restore the normal ability for the testes to respond to endogenous LH, which may be hindered for some time after the cycle is ended due to a prolonged state of inactivity. Once the HCG is stopped, the user continues treatment with Clomid alone. HCG should not be used for longer than two or three weeks though, as the resulting increased testosterone and estrogen levels may again initiate negative feedback inhibition at the hypothalamus. When planning your ancillary drug program, it is also important to remember that injectable steroids can stay active for a long duration. Using ancillary drugs the first week after a long acting injectable like Sustanon has been stopped may prove to be wholly ineffective. Instead, the athlete should wait for two to three weeks, to a point where androgen levels will be diminishing. Here the body will be primed and ready to restore testosterone production.

Clomid and HCG are also occasionally used periodically during a steroid cycle, in an effort to prevent natural testosterone levels from diminishing. In many instances this practice can prove difficult however, especially when using strong androgens for longer periods of time. There is also no exact method for using the two drugs in this manner. Some have experimented by periodically administering small doses of HCG along with one or two tablets of Clomid, perhaps for a few days at a stretch followed by a longer break. An on/off schedule would be implemented; for fear that this combination may lose some effectiveness if used continuously for this purpose. This method of intake may prove to be effective, although it is really much more feasible to stimulate testosterone production after the cycle than to try and maintain it for the long duration during.

In addition to helping with the post-cycle testosterone crash, this drug can also help with elevated estrogen levels during a steroid cycle. A high estrogen bevel puts an athlete in serious risk of developing gynecomastia, which is an obvious unwanted side effect. With the intake of Clomid, the athlete can hopefully reduce his risk for developing gynecomastia. The estrogen "blocking" properties of Clomid appear to be slightly weaker than Nolvadex in comparison however, which is why it is not usually thought of as an equal substitute for estrogen maintenance. Of course both drugs have similar actions in the body. and are relatively interchangeable for this purpose. Clomid can likewise also be used as a maintenance anti-estrogen throughout the duration of steroid cycle with good confidence, just as is done with Nolvadex. In most instances this will prove equally sufficient, the drug effectively minimizing the activity of estrogen in the body and warding off gyno and excess water/fat retention. Unfortunately just as with Nolvadex this is not always the case however, and many find it necessary to addition another anti-estrogenic drug. The most common adjunct is Proviron, an oral DHT used to competitively lower aromatase activity and raise the androgen to estrogen ratio. The Clomid/Nolvadex and Proviron combination is extremely effective, although we could alternately replace them both with a more specific aromatase inhibitor such as Arimidex,Femara, or Aromasin. While stronger at combating estrogen in most cases, these drugs are also typically much more costly.

As for toxicity and side effects, Clomid is considered a very safe drug. Bodybuilders seldom report any problems, but listed possible side effects do include hot flashes, nausea, dizziness, headaches and temporarily blurred vision. Such side effects usually only appear in females however, as they feel the effects of estrogen manipulation much more readily than men. While female athletes can clearly gain some benefit from this substance, estrogen manipulation is probably not the most comfortable way to go about cutting up. Should it still be used for such purposed and side effects do become pronounced, the drug of course is to be discontinued and (at least) a break taken from it.

Clomiphene citrate is widely available on the black market in a variety of brand names as well as generic tabs and liquid versions.

This product was the first multi-estered blend of testosterone available on the market, and was developed by Organon as an ideal HRT (Hormone Replacement Therapy) solution. To this day, most multi-estered blends are often referred to as the “(drug name) version of Sustanon” and many underground labs produce their own testosterone blends with a different number at the end (ex: “Sustanon 350” or whatever). It was meant to give the user consistently elevated blood levels of testosterone over the course of a month, and thus eliminate the need for frequent trips to the doctors office for injections.

This drug was very popular in the 80’s and 90’s, and was often thought of as being a “stack” in one drug. This isn’t really accurate, because, in the end all you have is testosterone once your body has metabolized it.Lately, it seems that this product has fallen out the radar for the most part, as it’s a bit expensive compared to other, single ester, testosterone products.Sustanon will do exactly what any other form of testosterone will do, no more and no less.

This means that it will cause muscle growth as well as fat loss, by sending a message to your muscles to store more protein (that’s its primary anabolic effect), while also protecting your muscles from catabolic (muscle wasting) (1). It will also change both the appearance and the actual number of muscle fibers in your body(2). We also find that aggression levels rise with the use of any form of testosterone (3), and it also increases erythropoiesis (red blood cell production) in your kidneys(4). This higher Red Blood Cell (RBC) count may actually improve endurance. Thus, it’s appropriate to say that Sustanon will increase strength, size, aggression, and maybe even endurance but certainly work capacity and recovery. More RBCs can also improve recovery from strenuous physical activity. In addition, Testosterone improves muscle contractile ability by two mechanisms: increasing the number of motor neutrons in muscle(5) and improving neuromuscular transmission(6). Also, testosterone is a well known promoter of glycogen synthesis.(7)As with any form of testosterone, Sustanon will convert to the primary female hormone estrogen (via a mechanism known as aromatization) when it is metabolized by the aromatize enzyme. Estrogen can lead to side effects, such as acne, the growth of breast tissue (called gynocomastia), as well as fat gain and reduced fat breakdown, testicular shrinkage and water retention. Testosterone is also metabolized into Dihydrotestosterone (DHT) by the 5alpha-reductase enzyme. This is a more androgenic form of Testosterone, and DHT has a high binding affinity to the tissues of both the prostate and the scalp resulting in hair loss in loss in users who may suffer from male pattern baldness. Prostate enlargement is also possible.

The only real downside to Sustanon is its relatively high cost. When compared with other forms of testosterone, you’re typically not getting a very good buy for your money when you choose to use any multi-estered product. However, as long as Sustanon remains easily accessible on the black market, it’s going to be found in cycles for a long time to come.References:1. J Lab Clin Med. 1995 Mar;125(3):326-33.2. Anat Histol Embryol. 2003 Apr;32(2):70-9.3. Health Psychol. 1990;9(6):774-91.4. Zhonghua Nan Ke Xue. 2003;9(4):248-515 Curr Opin Clin Nutr Metab Care. 2004 May;7(3):271-7.6. J Appl Physiol. 2001 Mar;90(3):850-6.7. Can J Physiol Pharmacol. 1999 Apr;77(4):300-4.